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BRCA1-deficient cancer cells benefit from DNA repair mechanism Malignancy cells are resourceful survivors with a lot of techniques for staying alive. Experts have uncovered among these stratagems, displaying how cells lacking the tumor suppressor BRCA1 can resume one type of DNA repair, sparing themselves from loss of life or stagnation. The study shows up in the January 21st problem of The Journal of Cell Biology. The BRCA1 protein really helps to mend double-strand DNA breaks by advertising homologous recombination. Without it, cells can amass damaged, jumbled, and fused chromosomes, which might cause them to end developing or die.